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There are also reports claiming that the amino acid leucine may be able to help control the effects of T3 on thyroid function: http://www.ncbi.nlm.nih.gov/pubmed/23664518
Both T3 and T4 act on thyroid hormones, TSH being the hormone that regulates thyroid production. (A recent study, however, has pointed to the fact that T3 does not have thyroid hormone action and is a poor choice as replacement therapy for thyroid malfunction.)
Leucine is also one amino acid that stimulates anabolic effects on skeletal muscle as well. It is the primary amino acid in muscle protein casein, causing it to rise in concentration as it is hydrolyzed. This means that it is a good source of amino acids that can raise lean body mass.
Athletes that have synthroid generic vs brand anabolic steroid use/decrease higher leucine levels (http://www.ncbi.nlm.nih.gov/pubmed/23274766). Where to buy viagra over the counter in brisbane
supplementation is also used to treat low T3, or the effects of chronic thyroid disease.
Leucine deficiency results in increased androgen levels. This is probably one reason for the higher increase/decrease in TSH athletes who are taking anabolic steroids.
Leucine Supplementation for T3 Control:
When leucine is raised with supplemental BCAAs, T3 stays high, and hyperinsulinaemic rats hyperthyroid humans are treated.
Leucine supplements Synthroid 25mcg $54 - $0.27 Per pill are also useful as a treatment for low T3. The research indicates that up to 40g leucine is needed provide a significant increase in T3, but the dose most needed to achieve this is not known. It also depends somewhat on age (younger people need more) and body weight.
The above leucine supplement would require between 100-200 grams of protein per day to meet the same requirements.
To sum up the effectiveness of leucine to regulate thyroid function.
Leucine has been used to control hyperthyroid symptoms with either T3 or T4.
A study is ongoing with leucine on T3 and T4. There was no change in TSH at the end of three months treatment.
A study with T3 and leucine (http://www.ncbi.nlm.nih.gov/pubmed/16485587) indicated that this treatment has not had a significant effect on the thyroid in hyperthyroid humans (no effects on TSH). Also, it appears that the higher dose needed to raise T3 has not made the hyperthyroid human benefit from leucine. This is the first treatment that seems to offer little benefit for hyperthyroids.
One study used leucine in combination with T4 to increase and provide a T3 replacement in hyperthyroid patients, and also had an effect on T4 (http://www.ncbi.nlm.nih.gov/pubmed/20503971). Interestingly, although they had sufficient levels of T4 at study entry, this group has a low TSH with the other therapies used, and no detectable T4.
A study using leucine alone in hyperthyroid patients did not increase TSH or thyroid volume.
A study using leucine in combination with T4 did not increase or thyroid volume at the end of three months, but this group is a high-risk group. The lower TSH has helped to increase thyroid volume, but the low T4 is probably contributing to this.
In addition, an increasing use of methylxanthines to control T3 has also included the use of leucine as a replacement. (http://www.ncbi.nlm.nih.gov/pubmed/15658074).
Although some individuals appear to respond leucine supplementation with lower hyperthyroid symptoms and thyroid function, it will require additional studies in that group to definitively determine if the leucine could have benefits for hyperthyroid patients.
T3 Replacement Therapy:
Most hyperthyroid patients take either methylxanthines, or T4 and TSH together.
Methylxanthines are a class of drugs that, like T3, can enhance thyroid production. However, T3 is better absorbed and less likely to cause hypothyroid states when taken in combination with methylxanthines.
A review of the literature over 20 years that included 500 patients showed T3 was more effective than T4 + TSH in improving thyroid function: http://.
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